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28th International Conference on Neurology and Cognitive Neuroscience, will be organized around the theme “Breakthroughs and Achievements in Neurocognitive Research”

Cognitive Neuroscience 2019 is comprised of keynote and speakers sessions on latest cutting edge research designed to offer comprehensive global discussions that address current issues in Cognitive Neuroscience 2019

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\r\n Cognitive neuroscience is the scientific field that is concerned with the study of the biological processes and aspects that underlie cognition, with a specific focus on the neural connections in the brain which are involved in mental processes.  It addresses the questions of how cognitive activities are affected or controlled by neural circuits in the brain. Cognitive neuroscience is a branch of both neuroscience and psychology, overlapping with disciplines such as physiological psychologycognitive psychology, and neuropsychology. Cognitive neuroscience relies upon theories in cognitive science coupled with evidence from neuropsychology, and computational modelling .

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  • Track 1-1Cognitive Psychology
  • Track 1-2Neuropsychology
  • Track 1-3Experimental Psychology
  • Track 1-4Physiological psychology
  • Track 1-5Cognitive genomics

 

<p style="\&quot;text-align:" justify;\"="">Dementia is usually caused by degeneration in the cerebral cortex, the part of the brain responsible for thoughts,     memories, actions, and personality. Death of brain cells in this region leads to the cognitive impairments that characterise dementia. Causes of dementia include head injury, brain tumours, infections, hormone disorders, metabolic disorders, hypoxia, nutritional deficiencies, drug abuse, or chronic alcoholism. Unfortunately, most disorders associated with dementia are progressive (inducing a gradual decline of functioning), degenerative (i.e. getting steadily worse over time), and irreversible. The two major degenerative causes of dementia are Alzheimer's disease (the progressive loss of nerve cells without known cause) and vascular dementia (i.e. loss of brain function due to a series of small strokes). Multiple neuropathologic processes may underlie dementia, including both neurodegenerative diseases and vascular disease.   Dementia is most common in elderly individuals, with advancing age being the strongest risk factor.

 

\r\n Vascular dementia is the broad term for dementia associated with problems of circulation of blood to the brain. Vascular brain injury (VBI) is widely recognized as a common cause of cognitive impairment (vascular cognitive impairment) culminating in vascular dementia. Most vascular dementia cases are sporadic and share risk factors with peripheral vascular disease. Vascular dementia is not a single disease; it is a group of syndromes relating to different vascular mechanisms.

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\r\n A Neurodegenerative disease includes Alzheimer's, Huntington's, Parkinson's, and the prion maladies, are genetically and pathologically linked to aberrant protein aggregation. AD, the most common type of dementia, is typically sporadic; however, rare mutations in several genes confer early onset. Release of the aggregation-prone peptides Aβ1–40 and Aβ1–42 by endoproteolysis of the amyloid precursor protein (APP) is associated with AD through an unknown mechanism that appears to be associated with Aβ aggregation. Typically, individuals who carry AD-linked mutations present with clinical symptoms during their fifth or sixth decade, while sporadic cases appear after the seventh decade. Although aggregation-mediated neurodegeneration emerges late in life, it is unclear whether this late onset is mechanistically linked to the aging process. The Pathophysiology of neurodegeneration is familial amyotrophic lateral sclerosis. Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder of the motor neurons in the spinal cord, brainstem, and motor cortex.

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\r\n Neuropsychiatry is a branch of medicine that deals with mental disorders attributable to diseases of the nervous system. It preceded the current disciplines of psychiatry and neurology.  However, psychiatry and neurology subsequently split apart and are typically practiced separately. Nevertheless, neuropsychiatry has become a growing subspecialty of psychiatry and it is also closely related to the fields of neuropsychology and Behavioral neurology, addresses clinical problems of cognition and/or behaviour caused by brain injury or brain disease . The division’s neuropsychiatry works collaboratively with the department’s neurologists to treat and manage the emotional and cognitive symptoms of neurological diseases. Such symptoms can include depression, anxiety, psychosis, hallucinations, and/or cognitive loss. Treatment can include psychotherapy and/or medication. Treating the psychiatric consequences of movement disorders allows patients to cultivate the best possible quality of life while also managing their medical issues.

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\r\n Neural dynamics is the study of the nervous system's remarkable capacity to change, and, at a systems level, the dynamic interplay between integration and segregation of brain regions enables all aspects of behaviour, including learning, memory, homeostasis and sensorimotor control. Real-time collision-free path planning and tracking control of a nonholonomic mobile robot in a dynamic environment is investigated using a neural dynamics based approach. The real-time robot path is generated through a dynamic neural activity landscape of a topologically organized neural network that represents the changing environment. The dynamics of each neuron is characterized by an additive neural dynamics model. The effectiveness and efficiency of this approach are demonstrated through simulation studies. A new approach for manipulating neural dynamics by using combined TMS and EEG recordings. Using the manipulative approach we can investigate (1) state-dependency in frequency-specific network connectivity by analyzing how TMS-evoked phase reset of on-going activity propagates from one cortical area to the rest of the brain in humans and (2) causal links between the neural dynamics and brain functions

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\r\n Neurological Nursing is a very challenging nursing specialty dealing with assessment, nursing diagnosis, and management of many neurological disorders for which nurses provide patient care. They are expected to work at health care clinics, hospitals and Intensive rehabilitation and brain injury units assisting complex Neuro surgeries, or with procedures in Neurointerventional Radiology and even in Clinic research areas. This includes trauma, brain injuries, stroke, seizures, tumours, headaches, infections, and aneurysms, as well as a host of other neurological complexities.

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\r\n It is increasingly recognised that pharmacological treatments for dementia should be used as a second-line approach and that non-pharmacological options should, in best practice, be pursued first. Non Pharmacological management include Standard therapies, Behavioural therapy, Reality orientation, Validation therapy etc. Reality orientation is one of the most widely used management strategies for dealing with people with dementia. t aims to help people with memory loss and disorientation by reminding them of facts about themselves and their environment. It can be used both with individuals and with groups. In either case, people with memory loss are oriented to their environment using a range of materials and activities. This involves consistent use of orientation devices such as signposts, notices and other memory aids.

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\r\n A drug used to treat neuropsychiatric, neuropsychological, or nervous-system disorders (as depression, obesity, schizophrenia, or Alzheimer's disease). Neuro-pharmaceutics focuses on identification of therapeutic targets, and then translating those discoveries into drug and therapy development. Research in neuropharmaceutics includes: drug discovery for limiting high frequency activity in epilepsy; drug transport proteins that control drug distribution to target tissues; spinal delivery of analgesics to chronic pain treatment; drugs for the treatment of depression, bipolar disorder, and schizophrenia; intranasal delivery methods for the treatment of Alzheimer’s disease; development of strategies to evaluate therapeutics on preclinical models of Alzheimer’s and Parkinson’s disease; anti-atherogenic and anti-diabetic therapies for the prevention and treatment of AD and other age-related dementias; and development of analgesic agents for chronic pain

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\r\n It is an older term for the treatment of disorders that affect the nervous system which psychological, psychiatric, and nervous disorders. Neurotherapeutics is a quarterly peer-reviewed medical journal covering research on experimental treatments of neurological disorders. There is a profound increase in the diagnostics procedure and drug discovery in the field of Neurology. It includes Stem cells and treatment, Nerve injury and repair Clinical Case report, Neurogenesis, cell and gene based approach and Neurotransmitter release and cell repair.

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\r\n The classic neuropathological signs of Cognitive disorders are amyloid plaques and neurofibrillary tangles. Plaques consist largely of the protein fragment beta-amyloid. This fragment is produced from a "parent" molecule called amyloid precursor protein. Tangles consist of tau, a protein normally involved in maintaining the internal structure of the nerve cell. While tau is normally modified by phosphorylation, or the attachment of phosphate molecules, excessive phosphorylation appears to contribute to tangle formation and prevents the protein from carrying out its normal functions. Oxidative stress, or damage to cellular structures by toxic oxygen molecules called free radicals, is also regarded as a pathology characteristic of Alzheimer’s. Individuals with Alzheimer’s typically experience brain inflammation. Many of the oldest patients with Alzheimer’s show signs of cerebrovascular disease in addition to "classic" Alzheimer's neuropathology.

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\r\n When nerve cells in the brain are damaged, they can no longer send information to each other in the normal way. This causes changes in the person's behaviour and abilities.

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\r\n Individuals with a moderate-to-severe brain injury often have problems in basic cognitive (thinking) skills such as paying attention, concentrating, and remembering new information and events. They may think slowly, speak slowly and solve problems slowly and confused easily when normal routines are changed or when things become too noisy or hectic around them. They may have speech and language problems, such as trouble finding the right word or understanding others. After brain injury, a person may have trouble with all the complex cognitive activities necessary to be independent and competent in our complex world. The brain processes large amounts of complex information all the time that allows us to function independently in our daily lives. This activity is called executive function because it means being the executive or being in charge of one's own life.

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\r\n After brain injury, a person may have trouble with all the complex cognitive activities necessary to be independent and competent in our complex world. The brain processes large amounts of complex information all the time that allows us to function independently in our daily lives. This activity is called executive function because it means being the executive or being in charge of one's own life which include Acting more dependent on others, Emotional or mood swings, Lack of motivation, Aggression, Lack of self-awareness

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Neurocognitive disorders are characterized by decline from an attained level of cognitive functioning mainly when physical changes occurs in brain, such as after neurological and mental illness, drug use, or brain injury. The two major degenerative causes of dementia are Alzheimer's disease and vascular dementia.  Dementia is usually caused by degeneration in the cerebral cortex, the part of the brain responsible for thoughts, memories, actions, and personality. Death of brain cells in this region leads to the cognitive impairments that characterise dementia. Vascular dementia is the broad term for dementia associated with problems of circulation of blood to the brain

 

  • Track 16-1Attention deficit hyperactive disorder
  • Track 16-2Hallucinogen persisting perception disorder
  • Track 16-3Mild cognitive impairment